Effects of Sodium Chlorate on Toxin Production by Escherichia coli O157:H7

Curr. Issues Intestinal Microbiol. (2004) 5: 19-22

Effects of Sodium Chlorate on Toxin Production by Escherichia coli O157:H7

T. R. Callaway, R. C. Anderson, T. S. Edrington, Y. S. Jung, K. M. Bischoff, K. J. Genovese, T. L. Poole, R. B. Harvey, J. A. Byrd and D. J. Nisbet

Chlorate kills E. coli O157:H7 and has been proposed as a feed additive to be included in cattle rations immediately prior to slaughter to reduce E. coli O157:H7 populations in the gut. Antibiotic usage is not recommended in cases of E. coli O157:H7-induced hemorrhagic colitis because some antibiotics stimulate increased toxin production. This study was undertaken to determine if chlorate treatment affected toxin production. Pure cultures of E. coli O157:H7 were treated with 1/4 MIC of antibiotics (ampicillin, tetracycline, ceftiofur, gentamicin, monensin, tylosin, penicillin, ciprofloxacin, and novobiocin); toxin production was significantly increased by some antibiotics, but not by chlorate. Studies with mixed fecal bacteria demonstrated that chlorate killed E. coli O157:H7, but again did not stimulate toxin production. Chlorate appears to be an effective method to reduce shiga toxin-producing E. coli (STEC) populations in food animals, but additional studies are warranted before it is used to control infections.

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